Epstein-Barr Virus Infection Alters Cellular Signal Cascades in Human Nasopharyngeal Epithelial Cells
Open Access
- 1 March 2006
- Vol. 8 (3) , 173-180
- https://doi.org/10.1593/neo.05625
Abstract
No abstract availableKeywords
This publication has 31 references indexed in Scilit:
- Stable expression of EBERs in immortalized nasopharyngeal epithelial cells confers resistance to apoptotic stressMolecular Carcinogenesis, 2005
- Epstein–Barr virus: 40 years onNature Reviews Cancer, 2004
- Epstein–Barr virus (EBV) LMP2A mediates B-lymphocyte survival through constitutive activation of the Ras/PI3K/Akt pathwayOncogene, 2004
- Phenotypic alterations induced by the Hong Kong‐prevalent Epstein‐Barr virus‐encoded LMP1 variant (2117‐LMP1) in nasopharyngeal epithelial cellsInternational Journal of Cancer, 2004
- A Positive Autoregulatory Loop of LMP1 Expression and STAT Activation in Epithelial Cells Latently Infected with Epstein-Barr VirusJournal of Virology, 2003
- Constitutive Stat3 activity up-regulates VEGF expression and tumor angiogenesisOncogene, 2002
- The Relationship Between Microvessel Density, the Expression of Vascular Endothelial Growth Factor (VEGF), and the Extension of Nasopharyngeal CarcinomaThe Laryngoscope, 2000
- Roles of STAT3 in mediating the cell growth, differentiation and survival signals relayed through the IL-6 family of cytokine receptorsOncogene, 2000
- STATs in oncogenesisOncogene, 2000
- Establishment of a cell line (NPC/HK1) from a differentiated squamous carcinoma of the nasopharynxInternational Journal of Cancer, 1980