Terminal Differentiation of Human Breast Cancer through PPARγ
- 1 February 1998
- journal article
- research article
- Published by Elsevier in Molecular Cell
- Vol. 1 (3) , 465-470
- https://doi.org/10.1016/s1097-2765(00)80047-7
Abstract
No abstract availableKeywords
This publication has 30 references indexed in Scilit:
- PPARγ induces cell cycle withdrawal: inhibition of E2F/DP DNA-binding activity via down-regulation of PP2AGenes & Development, 1997
- Inhibition of Adipogenesis Through MAP Kinase-Mediated Phosphorylation of PPARγScience, 1996
- Delayed Mammary Tumor Progression in Muc-1 Null MiceJournal of Biological Chemistry, 1995
- Sulfated Glycans Stimulate Endocytosis of the Cellular Isoform of the Prion Protein, PrPC, in Cultured CellsJournal of Biological Chemistry, 1995
- Stimulation of adipogenesis in fibroblasts by PPARγ2, a lipid-activated transcription factorCell, 1994
- mPPAR gamma 2: tissue-specific regulator of an adipocyte enhancer.Genes & Development, 1994
- Control of the peroxisomal β-oxidation pathway by a novel family of nuclear hormone receptorsPublished by Elsevier ,1992
- The PML-RARα fusion mRNA generated by the t(15;17) translocation in acute promyelocytic leukemia encodes a functionally altered RARCell, 1991
- Chromosomal translocation t(15;17) in human acute promyelocytic leukemia fuses RARα with a novel putative transcription factor, PMLCell, 1991
- A newly established metastatic breast tumor cell line with integrated amplified copies of ERBB2 and double minute chromosomesGenes, Chromosomes and Cancer, 1989