Genetic Defects of Apoptosis and Primary Immunodeficiency
- 31 May 2008
- journal article
- review article
- Published by Elsevier in Immunology and Allergy Clinics of North America
- Vol. 28 (2) , 329-351
- https://doi.org/10.1016/j.iac.2008.01.002
Abstract
No abstract availableKeywords
This publication has 103 references indexed in Scilit:
- B cells in HIV infection and diseaseNature Reviews Immunology, 2009
- Anti-HTLV antibody profiling reveals an antibody signature for HTLV-I-Associated Myelopathy/Tropical Spastic Paraparesis (HAM/TSP)Retrovirology, 2008
- Evidence for HIV-associated B cell exhaustion in a dysfunctional memory B cell compartment in HIV-infected viremic individualsThe Journal of Experimental Medicine, 2008
- Co-inherited mutations of Fas and caspase-10 in development of the autoimmune lymphoproliferative syndromeBMC Immunology, 2007
- Dominant inhibition of Fas ligand-mediated apoptosis due to a heterozygous mutation associated with autoimmune lymphoproliferative syndrome (ALPS) Type IbBMC Medical Genetics, 2007
- NRAS mutation causes a human autoimmune lymphoproliferative syndromeProceedings of the National Academy of Sciences, 2007
- Elimination of Antigen-Presenting Cells and Autoreactive T Cells by Fas Contributes to Prevention of AutoimmunityImmunity, 2007
- Rapid antibody quantification and generation of whole proteome antibody response profiles using LIPS (luciferase immunoprecipitation systems)Biochemical and Biophysical Research Communications, 2007
- Rapamycin improves lymphoproliferative disease in murine autoimmune lymphoproliferative syndrome (ALPS)Blood, 2006
- Abnormal disulfide-linked oligomerization results in ER retention and altered signaling by TNFR1 mutants in TNFR1-associated periodic fever syndrome (TRAPS)Blood, 2006