Receptor mechanisms and circuitry underlying NMDA antagonist neurotoxicity
- 1 January 2002
- journal article
- research article
- Published by Springer Nature in Molecular Psychiatry
- Vol. 7 (1) , 32-43
- https://doi.org/10.1038/sj.mp.4000912
Abstract
Molecular Psychiatry publishes work aimed at elucidating biological mechanisms underlying psychiatric disorders and their treatmentKeywords
This publication has 65 references indexed in Scilit:
- Disseminated Corticolimbic Neuronal Degeneration Induced in Rat Brain by MK-801: Potential Relevance to Alzheimer's DiseaseNeurobiology of Disease, 1998
- Safety and Tolerability of the Glutamate Antagonist CGS 19755 (Selfotel) in Patients With Acute Ischemic StrokeStroke, 1995
- Neuronal Vacuolization and Necrosis Induced by the Noncompetitive N-methyl-d-aspartate (NMDA) Antagonist MK(+)801 (Dizocilpine Maleate): A Light and Electron Microscopic Evaluation of the Rat Retrosplenial CortexExperimental Neurology, 1993
- MK-801 increases endogenous acetylcholine release in the rat parietal cortex: a study using brain microdialysisNeuroscience Letters, 1993
- NMDA Antagonist Neurotoxicity: Mechanism and PreventionScience, 1991
- The Influence of Ketamine on Regional Brain Glucose UseAnesthesiology, 1988
- Cholinergic innervation of cortex by the basal forebrain: Cytochemistry and cortical connections of the septal area, diagonal band nuclei, nucleus basalis (Substantia innominata), and hypothalamus in the rhesus monkeyJournal of Comparative Neurology, 1983
- Local Changes in Cerebral Glucose Utilization during Ketamine AnesthesiaAnesthesiology, 1982
- Localisation of phencyclidine-induced changes in brain energy metabolismNature, 1979
- Measurement of regional brain glucose utilization in vivo using [2(-14)C] glucose.Stroke, 1979