Glutathione Status of Isolated Rat Hepatocytes Affects Bile Acid-Induced Cellular Necrosis But Not Apoptosis
- 1 April 2000
- journal article
- Published by Elsevier in Toxicology and Applied Pharmacology
- Vol. 164 (1) , 102-111
- https://doi.org/10.1006/taap.2000.8894
Abstract
No abstract availableKeywords
This publication has 51 references indexed in Scilit:
- CD95-mediated murine hepatic apoptosis requires an intact glutathione statusHepatology, 1999
- Rapid and Specific Efflux of Glutathione before Hepatocyte Injury Induced by HypoxiaBiochemical and Biophysical Research Communications, 1997
- Glutathione depletion in fibroblasts is the basis for apoptosis-induction by endogenous reactive oxygen speciesCell Death & Differentiation, 1997
- Mitochondrial control of apoptosisPublished by Elsevier ,1997
- Non-oxidative Loss of Glutathione in Apoptosis via GSH ExtrusionBiochemical and Biophysical Research Communications, 1995
- No requirement of reactive oxygen intermediates in Fas‐mediated apoptosisFEBS Letters, 1994
- Insulin and glucocorticoid dependence of hepatic gamma-glutamylcysteine synthetase and glutathione synthesis in the rat. Studies in cultured hepatocytes and in vivo.Journal of Clinical Investigation, 1992
- 1-Bromoalkanes as new potent nontoxic glutathione depletors in isolated rat hepatocytesBiochemical and Biophysical Research Communications, 1991
- Effect of chenodeoxycholic and ursodeoxycholic acids on isolated adult human hepatocytesDigestive Diseases and Sciences, 1984
- The inhibition of γ-glutamyl transpeptidase and glutathione metabolism of isolated rat kidney cells by L-(αS, 5S)-α-amino-3-chloro-4, 5-dihydro-5-isoxazoleacetic acid (AT-125; NSC-163501)Biochemical and Biophysical Research Communications, 1980