Nicotine-induced Fos expression in the nucleus accumbens and the medial prefrontal cortex of the rat: Role of nicotinic and NMDA receptors in the ventral tegmental area
- 27 April 2000
- Vol. 36 (4) , 314-321
- https://doi.org/10.1002/(sici)1098-2396(20000615)36:4<314::aid-syn8>3.0.co;2-u
Abstract
No abstract availableKeywords
This publication has 33 references indexed in Scilit:
- Molecular Adaptations to Psychostimulants in Striatal Neurons: Toward a Pathophysiology of AddictionNeurobiology of Disease, 1997
- Burst stimulation of the medial forebrain bundle selectively increases Fos-like immunoreactivity in the limbic forebrain of the ratNeuroscience, 1996
- Tonic Activation of NMDA Receptors Causes Spontaneous Burst Discharge of Rat Midbrain Dopamine Neurons In VivoEuropean Journal of Neuroscience, 1993
- Regulation of immediate early gene expression and AP-1 binding in the rat nucleus accumbens by chronic cocaine.Proceedings of the National Academy of Sciences, 1992
- Burst firing of mesencephalic dopamine neurons is inhibited by somatodendritic application of kynurenateActa Physiologica Scandinavica, 1991
- Amphetamine and cocaine induce drug-specific activation of the c-fos gene in striosome-matrix compartments and limbic subdivisions of the striatum.Proceedings of the National Academy of Sciences, 1990
- Lack of tolerance to nicotine-induced dopamine release in the nucleus accumbensEuropean Journal of Pharmacology, 1989
- The excitatory amino acid antagonist kynurenate induces pacemaker‐like firing of dopamine neurons in rat ventral tegmental area in vivoActa Physiologica Scandinavica, 1988
- Nicotine preferentially stimulates dopamine release in the limbic system of freely moving ratsEuropean Journal of Pharmacology, 1986
- Nicotinic effects on the firing pattern of midbrain dopamine neuronsActa Physiologica Scandinavica, 1986