ANTICONVULSANT INDUCED INCREASE IN 25-HYDROXY-VITAMIN D3-lα-HYDROXYLASE

Abstract
Osteomalacia is associated with long-term anticonvulsant therapy [in humans]. Anticonvulsants modify the hepatic metabolism of vitamin D3 and decrease serum 25-hydroxyvitamin D3 (25-OH-D3] levels. Diphenylhydantoin (DPH) and phenobarbitone (PB) enhance the activity of kidney 25-hydroxy-vitamin D3-1.alpha.-hydroxylase (1-hydroxylase) in the chicken. Anticonvulsant osteomalacia may not be due to a lack of the active metabolite of vitamin D, 1,25-dihydroxyvitamin D3 (1,25(OH)2D3).

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