Loss of Smad4 Function in Pancreatic Tumors
Open Access
- 1 November 2001
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 276 (46) , 43175-43181
- https://doi.org/10.1074/jbc.m105895200
Abstract
No abstract availableKeywords
This publication has 46 references indexed in Scilit:
- Distinct Oligomeric States of SMAD Proteins in the Transforming Growth Factor-β PathwayJournal of Biological Chemistry, 2000
- Transforming Growth Factor β-Independent Shuttling of Smad4 between the Cytoplasm and NucleusMolecular and Cellular Biology, 2000
- TGFβ Signaling in Growth Control, Cancer, and Heritable DisordersCell, 2000
- Importin β Mediates Nuclear Translocation of Smad 3Journal of Biological Chemistry, 2000
- Immunohistochemical Labeling for Dpc4 Mirrors Genetic Status in Pancreatic AdenocarcinomasThe American Journal of Pathology, 2000
- THE UBIQUITIN SYSTEMAnnual Review of Biochemistry, 1998
- Dual role of the Smad4/DPC4 tumor suppressor in TGFβ-inducible transcriptional complexesGenes & Development, 1997
- Partnership between DPC4 and SMAD proteins in TGF-β signalling pathwaysNature, 1996
- DPC4 , A Candidate Tumor Suppressor Gene at Human Chromosome 18q21.1Science, 1996
- The POZ domain: a conserved protein-protein interaction motif.Genes & Development, 1994