Disruption of the Lecithin:Retinol Acyltransferase Gene Makes Mice More Susceptible to Vitamin A Deficiency

This publication has 61 references indexed in Scilit:

• c-Jun N-Terminal Kinase Contributes to Aberrant Retinoid Signaling in Lung Cancer Cells by Phosphorylating and Inducing Proteasomal Degradation of Retinoic Acid Receptor α
  Molecular and Cellular Biology, 2005
• Expression profiles of Id1 and p16 proteins in all-trans-retinoic acid-induced apoptosis and cell cycle re-distribution in melanoma
  Cancer Letters, 2005
• RETINOIC ACID RECEPTORS AND CANCERS
  Annual Review of Nutrition, 2004
• Retinoic acid causes cell growth arrest and an increase in p27 in F9 wild type but not in F9 retinoic acid receptor β2 knockout cells
  Experimental Cell Research, 2004
• Lecithin-retinol Acyltransferase Is Essential for Accumulation of All-trans-Retinyl Esters in the Eye and in the Liver
  Journal of Biological Chemistry, 2004
• Distinct Retinoid Metabolic Functions for Alcohol Dehydrogenase Genes Adh1 and Adh4 in Protection against Vitamin A Toxicity or Deficiency Revealed in Double Null Mutant Mice
  Journal of Biological Chemistry, 2002
• A Hoxa2 knockin allele that expresses EGFP upon conditional Cre‐mediated recombination
  Genesis, 2002
• Spermiogenesis Deficiency in Mice Lacking the Trf2 Gene
  Science, 2001
• Biochemical Pathways of Retinoid Transport, Metabolism, and Signal Transduction
  Clinical Immunology and Immunopathology, 1996
• Vitamin A Storage in Lungs during Perinatal Development in the Rat
  Neonatology, 1990