ER Stress Regulation of ATF6 Localization by Dissociation of BiP/GRP78 Binding and Unmasking of Golgi Localization Signals
Top Cited Papers
- 1 July 2002
- journal article
- Published by Elsevier in Developmental Cell
- Vol. 3 (1) , 99-111
- https://doi.org/10.1016/s1534-5807(02)00203-4
Abstract
No abstract availableKeywords
Funding Information
- National Institutes of Health (CA50329)
This publication has 50 references indexed in Scilit:
- The Luminal Domain of ATF6 Senses Endoplasmic Reticulum (ER) Stress and Causes Translocation of ATF6 from the ER to the GolgiJournal of Biological Chemistry, 2002
- IRE1 couples endoplasmic reticulum load to secretory capacity by processing the XBP-1 mRNANature, 2002
- XBP1 mRNA Is Induced by ATF6 and Spliced by IRE1 in Response to ER Stress to Produce a Highly Active Transcription FactorCell, 2001
- Identification of the G13 (cAMP-response-element-binding protein-related protein) gene product related to activating transcription factor 6 as a transcriptional activator of the mammalian unfolded protein responseBiochemical Journal, 2001
- Regulated Intramembrane ProteolysisCell, 2000
- Setting the Standards: Quality Control in the Secretory PathwayScience, 1999
- Transport-Dependent Proteolysis of SREBPCell, 1999
- Characterization of the Nucleotide Binding Properties and ATPase Activity of Recombinant Hamster BiP Purified from BacteriaJournal of Biological Chemistry, 1995
- In Vitro Dissociation of BiP-Peptide Complexes Requires a Conformational Change in BiP after ATP Binding but Does Not Require ATP HydrolysisJournal of Biological Chemistry, 1995
- Posttranslational association of immunoglobulin heavy chain binding protein with nascent heavy chains in nonsecreting and secreting hybridomas.The Journal of cell biology, 1986