SNO Is a Probable Target for Gene Amplification at 3q26 in Squamous-Cell Carcinomas of the Esophagus

This publication has 35 references indexed in Scilit:

• Nonrandom Chromosomal Imbalances in Esophageal Squamous Cell Carcinoma Cell Lines: Possible Involvement of the ATF3 and CENPF Genes in the 1q32 Amplicon
  Japanese Journal of Cancer Research, 2000
• Comparative Genomic Hybridization Analysis Detects Frequent Over-Representation of DNA Sequences at 3q, 7p, and 8q in Head and Neck Carcinomas
  Cancer Genetics and Cytogenetics, 2000
• Molecular Definition of a Small Amplification Domain within 3q26 in Tumors of Cervix, Ovary, and Lung
  Cancer Genetics and Cytogenetics, 2000
• Oncogene amplification in solid tumors
  Seminars in Cancer Biology, 1999
• Molecular cytogenetic fingerprinting of esophageal squamous cell carcinoma by comparative genomic hybridization reveals a consistent pattern of chromosomal alterations
  Genes, Chromosomes and Cancer, 1999
• Comparative genomic hybridization of squamous cell carcinoma of the esophagus: The possible involvement of theDP1 gene in the 13q34 amplicon
  Genes, Chromosomes and Cancer, 1999
• Expression analysis of genes at 3q26-q27 involved in frequent amplification in squamous cell lung carcinoma
  European Journal Of Cancer, 1999
• Comparative genomic hybridization detects frequent overrepresentation of chromosomal material from 3q26, 8q24, and 20q13 in human ovarian carcinomas
  Genes, Chromosomes and Cancer, 1997
• Gain of chromosome 3q defines the transition from severe dysplasia to invasive carcinoma of the uterine cervix.
  Proceedings of the National Academy of Sciences, 1996
• Amplification of the EGF receptor and c‐myc genes in human esophageal cancers
  International Journal of Cancer, 1988