Anti-Fas Induces Hepatic Chemokines and Promotes Inflammation by an NF-κB-independent, Caspase-3-dependent Pathway
Open Access
- 1 December 2001
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 276 (52) , 49077-49082
- https://doi.org/10.1074/jbc.m109791200
Abstract
No abstract availableKeywords
This publication has 44 references indexed in Scilit:
- Macrophages that have ingested apoptotic cells in vitro inhibit proinflammatory cytokine production through autocrine/paracrine mechanisms involving TGF-beta, PGE2, and PAF.Journal of Clinical Investigation, 1998
- Selective activation of the JNK/AP‐1 pathway in FAS‐mediated apoptosis of rheumatoid arthritis synoviocytesArthritis & Rheumatism, 1997
- Human Chemokines: An UpdateAnnual Review of Immunology, 1997
- MAP3K-related kinase involved in NF-KB induction by TNF, CD95 and IL-1Nature, 1997
- Apoptosis by Death FactorCell, 1997
- Fas-Mediated Stimulation Induces IL-8 Secretion by Rheumatoid Arthritis Synoviocytes Independently of CPP32-Mediated ApoptosisBiochemical and Biophysical Research Communications, 1996
- The CD95 (APO-1/Fas) Receptor Activates NF- B Independently of Its Cytotoxic FunctionPublished by Elsevier ,1996
- Lethal effect of the anti-Fas antibody in miceNature, 1993
- Phagocyte recognition of cells undergoing apoptosisImmunology Today, 1993
- Rat KC cDNA cloning and mRNA expression in lung macrophages and fibroblastsBiochemical and Biophysical Research Communications, 1992