Bradykinin release of TNF-α plays a key role in the development of inflammatory hyperalgesia
- 1 June 1993
- journal article
- neurogenic pain-and-inflammation
- Published by Springer Nature in Inflammation Research
- Vol. 38 (2) , C7-C9
- https://doi.org/10.1007/bf01991120
Abstract
No abstract availableKeywords
This publication has 7 references indexed in Scilit:
- The pivotal role of tumour necrosis factor α in the development of inflammatory hyperalgesiaBritish Journal of Pharmacology, 1992
- Interleukin‐8 as a mediator of sympathetic painBritish Journal of Pharmacology, 1991
- Bradykinin stimulates tumor necrosis factor and interleukin‐1 release from macrophagesFEBS Letters, 1989
- Interleukin-1β as a potent hyperalgesic agent antagonized by a tripeptide analogueNature, 1988
- A peripheral sympathetic component in inflammatory hyperalgesiaEuropean Journal of Pharmacology, 1987
- Interleukin-1 and the Pathogenesis of the Acute-Phase ResponseNew England Journal of Medicine, 1984
- Central and peripheral antialgesic action of aspirin-like drugsEuropean Journal of Pharmacology, 1978