Sodium channels as molecular targets for antiepileptic drugs
- 1 March 1998
- journal article
- review article
- Published by Elsevier in Brain Research Reviews
- Vol. 26 (1) , 16-28
- https://doi.org/10.1016/s0165-0173(97)00054-4
Abstract
No abstract availableKeywords
This publication has 94 references indexed in Scilit:
- Anticonvulsant activity of topiramate and phenytoin in a rat model of ischemia-induced epilepsyLife Sciences, 1996
- Functional Co-expression of the β1 and Type IIA α Subunits of Sodium Channels in a Mammalian Cell LinePublished by Elsevier ,1995
- Na+ channels must deactivate to recover from inactivationNeuron, 1994
- Localization of a putative human brain sodium channel gene (SCN1A) to chromosome band 2q24Cytogenetic and Genome Research, 1994
- Lamotrigine as an Add-On Drug in the Management of Lennox-Gastaut SyndromeEuropean Neurology, 1992
- Three brain sodium channel α-subunit genes are clustered on the proximal segment of mouse chromosome 2Genomics, 1991
- Inactivation of cloned Na channels expressed in Xenopus oocytes.The Journal of general physiology, 1990
- Localization of a human brain sodium channel gene (SCN2A) to chromosome 2Genomics, 1989
- Primary structure of rat brain sodium channel III deduced from the cDNA sequenceFEBS Letters, 1988
- Time- and voltage-dependent interactions of antiarrhythmic drugs with cardiac sodium channelsBiochimica et Biophysica Acta (BBA) - Reviews on Biomembranes, 1977