Neuropsychological Impairment From Acute Low-Level Exposure to Carbon Monoxide

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Abstract
CARBON MONOXIDE poisoning results primarily in cardiac and neurologic damage.1 The severity of carbon monoxide toxicity ranges from subclinical or occult poisoning to severe morbidity and death. The effect of low-level exposure to carbon monoxide on the heart is well documented. In patients with coronary heart disease, inhalation of slightly elevated concentrations of carbon monoxide in air produces mean blood carboxyhemoglobin (HbCO) levels of 0.02 to 0.04 and causes coronary ischemia.2 Carboxyhemoglobin levels of 0.06 have been reported to precipitate cardiac arrhythmias.3

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