Different amyloidogenic peptides share a similar mechanism of neurotoxicity involving reactive oxygen species and calcium
- 6 April 1995
- journal article
- research article
- Published by Elsevier in Brain Research
- Vol. 676 (1) , 219-224
- https://doi.org/10.1016/0006-8993(95)00148-j
Abstract
No abstract availableKeywords
This publication has 26 references indexed in Scilit:
- β-Amyloid Peptide Free Radical Fragments Initiate Synaptosomal Lipoperoxidation in a Sequence-Specific Fashion: Implications to Alzheimer′s DiseaseBiochemical and Biophysical Research Communications, 1994
- Soluble β-Amyloid Induction of Alzheimer's Phenotype for Human Fibroblast K + ChannelsScience, 1994
- Genetics and molecular advances in Alzheimer's diseaseTrends in Neurosciences, 1993
- Physiological production of the β-amyloid protein and the mechanism of Alzheimer's diseaseTrends in Neurosciences, 1993
- Calcium-destabilizing and neurodegenerative effects of aggregated β-amyloid peptide are attenuated by basic FGFBrain Research, 1993
- β-Amyloid Protein Amplifies Calcium Signaling in Central Neurons from the Adult MouseBiochemical and Biophysical Research Communications, 1993
- Release of Excess Amyloid β Protein from a Mutant Amyloid β Protein PrecursorScience, 1993
- AmylinLife Sciences, 1992
- B- Amyloid increases neuronal susceptibility to injufy by glucose deprivationNeuroReport, 1991
- Neurotrophic and Neurotoxic Effects of Amyloid β Protein: Reversal by Tachykinin NeuropeptidesScience, 1990