THYROID-ACTIVITY DURING HEPATOCARCINOGENESIS BY N-2-FLUORENYLACETAMIDE IN THE MALE-RAT
- 1 January 1982
- journal article
- research article
- Vol. 36 (1) , 105-117
Abstract
During chemical hepatocarcinogenesis by N-2-fluorenylacetamide, the hormonal status of male Sprague Dawley rats is modified. The histological study of the thyroid gland demonstrates a decreased activity evidenced by an augmentation of the follicle size and a reduced size of follicular epithelium. The levels of T3 [triiodothyronine] and T4 [thyroxine] reflect in part this decreased activity. Comparison with female rats treated in the same conditions shows an opposite effect of N-2-fluorenylacetamide. In the female the thyroid gland presented a hyperactive state accompanied by a drastic decrease of serum T3 and T4. This difference in thyroid activity might play an important part in the mechanism responsible for the sex difference in liver cancer induction by N-2-fluorenylacetamide.This publication has 4 references indexed in Scilit:
- INHIBITION OF LOCAL AND METASTATIC HEPATOMA GROWTH AND PROLONGATION OF SURVIVAL AFTER INDUCTION OF HYPOTHYROIDISM1981
- TRIIODOTHYRONINE - ON ITS ROLE AS A SPECIFIC HEPATOMITOGEN1980
- Inhibition of the growth of Morris Hepatoma #44 in rats after induction of hypothyroidism: Evidence that Morris Hepatomas are thyroid dependentGastroenterology, 1979
- THYROID-HORMONE IN 7,12-DIMETHYLBENZ(A)ANTHRACENE-INDUCED LEUKEMIA IN RATS1979