Infusion of recombinant human acid sphingomyelinase into Niemann‐Pick disease mice leads to visceral, but not neurological, correction of the pathophysiology

Abstract
An inherited deficiency of acid sphingomyelinase (ASM) activity results in the Type A and B forms of Niemann-Pick disease (NPD). Using the ASM-deficient mouse model (ASMKO) of NPD, we evaluated the e...
Funding Information
  • National Institutes of Health (HD 28607, HD 32654)
  • United States - Israel Binational Science Foundation (93-00015)
  • National Center for Research Resources (RR 0071)