Nicorandil, a potent cardioprotective agent, acts by opening mitochondrial ATP-dependent potassium channels
Open Access
- 27 January 2000
- journal article
- research article
- Published by Elsevier in Journal of the American College of Cardiology
- Vol. 35 (2) , 514-518
- https://doi.org/10.1016/s0735-1097(99)00552-5
Abstract
No abstract availableKeywords
This publication has 25 references indexed in Scilit:
- Intravenous nicorandil can preserve microvascular integrity and myocardial viability in patients with reperfused anterior wall myocardial infarctionJournal of the American College of Cardiology, 1999
- Myocardial Distribution and Biotransformation In Vitro and In Vivo of Nicorandil in Rats, with Special Reference to MitochondriaJournal of Cardiovascular Pharmacology, 1999
- Cardioprotection by opening of the KATPchannel in unstable angina Is this a clinical manifestation of myocardial preconditioning? Results of a randomized study with nicorandilPublished by Oxford University Press (OUP) ,1999
- Modulation of Mitochondrial ATP-Dependent K + Channels by Protein Kinase CCirculation Research, 1998
- Mitochondrial ATP-Dependent Potassium ChannelsCirculation, 1998
- Myocardial Protection Afforded by Nicorandil and Ischaemic Preconditioning in a Rabbit Infarct Model In VivoJournal of Cardiovascular Pharmacology, 1998
- Cardioprotective Effect of Intravenous Nicorandil in Patients With Successful Reperfusion for Acute Myocardial InfarctionJapanese Circulation Journal, 1998
- Cardioprotective Effect of Diazoxide and Its Interaction With Mitochondrial ATP-Sensitive K + ChannelsCirculation Research, 1997
- Synergistic Modulation of ATP-Sensitive K + Currents by Protein Kinase C and AdenosineCirculation Research, 1996
- Nicorandil as a hybrid between nitrates and potassium channel activatorsThe American Journal of Cardiology, 1989