Nerve Terminal GABAA Receptors Activate Ca2+/Calmodulin-dependent Signaling to Inhibit Voltage-gated Ca2+ Influx and Glutamate Release

This publication has 99 references indexed in Scilit:

• GABAA receptor trafficking and its role in the dynamic modulation of neuronal inhibition
  Nature Reviews Neuroscience, 2008
• Presynaptic GABA_A receptors facilitate GABAergic transmission to dopaminergic neurons in the ventral tegmental area of young rats
  The Journal of Physiology, 2007
• Activation of presynaptic GABAA receptors increases spontaneous glutamate release onto noradrenergic neurons of the rat locus coeruleus
  Brain Research, 2005
• Variations on an inhibitory theme: phasic and tonic activation of GABAA receptors
  Nature Reviews Neuroscience, 2005
• Disturbances in Glucose-Tolerance, Insulin-Release, and Stress-Induced Hyperglycemia upon Disruption of the Cav2.3 ( 1E) Subunit of Voltage-Gated Ca2+ Channels
  Molecular Endocrinology, 2002
• Presynaptic GABA_B Receptor Modulation of Glutamate Exocytosis from Rat Cerebrocortical Nerve Terminals: Receptor Decoupling by Protein Kinase C
  Journal of Neurochemistry, 1998
• GABA-Activated Chloride Channels in Secretory Nerve Endings
  Science, 1993
• 4‐Aminobutyrate Can Be Released Exocytotically from Guinea‐Pig Cerebral Cortical Synaptosomes
  Journal of Neurochemistry, 1987
• V. Barbiturate and benzodiazepine modulation of GABA receptor binding and function
  Life Sciences, 1986
• Influx of calcium, strontium, and barium in presynaptic nerve endings.
  The Journal of general physiology, 1982