S-Allylcysteine prevents amyloid-β peptide-induced oxidative stress in rat hippocampus and ameliorates learning deficits
- 12 April 2004
- journal article
- research article
- Published by Elsevier in European Journal of Pharmacology
- Vol. 489 (3) , 197-202
- https://doi.org/10.1016/j.ejphar.2004.03.001
Abstract
No abstract availableKeywords
This publication has 25 references indexed in Scilit:
- Overexpression of superoxide dismutase 1 protects against β-amyloid peptide toxicity: effect of estrogen and copper chelatorsNeurochemistry International, 2004
- Neurotoxicity induced by amyloid β-peptide and ibotenic acid in organotypic hippocampal cultures: protection by S-allyl-l-cysteine, a garlic compoundBrain Research, 2003
- Anti-apoptotic proteins are oxidized by Aβ25–35 in Alzheimer's fibroblastsBiochimica et Biophysica Acta (BBA) - Molecular Basis of Disease, 2003
- Antioxidant Treatment in Alzheimer's Disease: Current StateJournal of Molecular Neuroscience, 2003
- Natural extracts as possible protective agents of brain agingNeurobiology of Aging, 2002
- Acetyl-l-Carnitine Protects Against Amyloid-Beta Neurotoxicity: Roles of Oxidative Buffering and ATP LevelsNeurochemical Research, 2002
- Neuroprotective and neurorescuing effects of isoform-specific nitric oxide synthase inhibitors, nitric oxide scavenger, and antioxidant against beta-amyloid toxicityBritish Journal of Pharmacology, 2001
- Differential regulation of NO availability from macrophages and endothelial cells by the garlic component S-allyl cysteineFree Radical Biology & Medicine, 2001
- Garlic Compounds Minimize Intracellular Oxidative Stress and Inhibit Nuclear Factor-κB ActivationJournal of Nutrition, 2001
- S-Allyl Cysteine Inhibits Activation of Nuclear Factor Kappa B in Human T CellsFree Radical Biology & Medicine, 1997