Role of Opioid δ1Receptors, Mitochondrial KATPChannels, and Protein Kinase C during Cardiocyte Apoptosis
- 1 November 2001
- journal article
- retracted article
- Published by Elsevier in Journal of Molecular and Cellular Cardiology
- Vol. 33 (11) , 2007-2014
- https://doi.org/10.1006/jmcc.2001.1464
Abstract
No abstract availableKeywords
This publication has 24 references indexed in Scilit:
- Unresolved Issues Regarding the Role of Apoptosis in the Pathogenesis of Ischemic Injury and Heart FailureJournal of Molecular and Cellular Cardiology, 2000
- Apoptosis in cardiac diseaseThe American Journal of Medicine, 1999
- Direct Preconditioning of Cardiac Myocytes via Opioid Receptors and K ATP ChannelsCirculation Research, 1999
- Ischemic Preconditioning Decreases Apoptosis in Rat Hearts In VivoCirculation, 1997
- Apoptosis in Human Acute Myocardial InfarctionCirculation, 1997
- Acute Myocardial Infarction in Humans is Associated with Activation of Programmed Myocyte Cell Death in the Surviving Portion of the HeartJournal of Molecular and Cellular Cardiology, 1996
- Naloxone Blockade of Myocardial Ischemic Preconditioning is StereoselectiveJournal of Molecular and Cellular Cardiology, 1996
- Reperfusion injury induces apoptosis in rabbit cardiomyocytes.Journal of Clinical Investigation, 1994
- ATP-sensitive K+ channel in the mitochondrial inner membraneNature, 1991
- ATP-regulated K+ channels in cardiac muscleNature, 1983