Abstract
The author discusses the role of the following three factors in the pathogenesis of central serous chorioretinopathy: intraocular tension, adhesion of the pigment epithelium and choroidal hydrostatic pressure. Some cases of central serous chorioretinopathy present the angiographic findings typical of retinal pigment epitheliitis, others show choroidal perfusion defects in the area of subretinal leakage. Both are to be considered as secondary forms and distinguished from the idiopathic disease.

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