Herbimycin A and Geldanamycin Inhibit Okadaic Acid-Induced Apoptosis and p38 Activation in NRK-52E Renal Epithelial Cells
- 15 November 1999
- journal article
- Published by Elsevier in Toxicology and Applied Pharmacology
- Vol. 161 (1) , 59-74
- https://doi.org/10.1006/taap.1999.8765
Abstract
No abstract availableKeywords
This publication has 37 references indexed in Scilit:
- Activation Mechanism of the MAP Kinase ERK2 by Dual PhosphorylationCell, 1997
- The Role of c-Jun N-terminal Kinase (JNK) in Apoptosis Induced by Ultraviolet C and γ RadiationJournal of Biological Chemistry, 1996
- Differential sensitivity of normal and H-ras oncogene-transformed ratkidney epithelial cells to okadaic acid-induced apoptosisToxicology and Applied Pharmacology, 1996
- Structural Analysis of the MAP Kinase ERK2 and Studies of MAP Kinase Regulatory PathwaysPublished by Elsevier ,1996
- Mechanoreception at the cellular level: the detection, interpretation, and diversity of responses to mechanical signalsBiochemistry and Cell Biology, 1995
- Parallel signal processing among mammalian MAPKsTrends in Biochemical Sciences, 1995
- Characterization of a Renal Epithelial Cell Model of Apoptosis Using Okadaic Acid and the NRK-52E Cell LineToxicologic Pathology, 1994
- JNK1: A protein kinase stimulated by UV light and Ha-Ras that binds and phosphorylates the c-Jun activation domainPublished by Elsevier ,1994
- Critical tyrosine residues regulate the enzymatic and biological activity of Raf-1 kinase.Molecular and Cellular Biology, 1993
- Okadaic acid: a new probe for the study of cellular regulationTrends in Biochemical Sciences, 1990