Abstract
Microinjections of β-alanine and glycine into the nucleus tractus solitarii (NTS) of the rat, led to an increase in blood pressure and heart rate. The responses to glycine but not to β-alanine could be blocked by strychnine. Both amino acids reduced the responses to aortic nerve stimulation. High K+ stimulation caused a calcium-dependent release of β-alanine from tissues in the area of the NTS. These results suggest that β-alanine and glycine may modulate the cardiovascular control within the NTS.

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