Ras-Guanine Nucleotide-Releasing Factor 1 (Ras-GRF1) Controls Activation of Extracellular Signal-Regulated Kinase (ERK) Signaling in the Striatum and Long-Term Behavioral Responses to Cocaine
- 1 October 2009
- journal article
- Published by Elsevier in Biological Psychiatry
- Vol. 66 (8) , 758-768
- https://doi.org/10.1016/j.biopsych.2009.03.014
Abstract
No abstract availableKeywords
This publication has 90 references indexed in Scilit:
- Epigenetic regulation in psychiatric disordersNature Reviews Neuroscience, 2007
- Laser microdissection and microarray analysis of the hippocampus of Ras-GRF1 knockout mice reveals gene expression changes affecting signal transduction pathways related to memory and learningNeuroscience, 2007
- ERK2: a logical AND gate critical for drug-induced plasticity?Current Opinion in Pharmacology, 2007
- Dual alteration of limbic dopamine D1receptor-mediated signalling and the Akt/GSK3 pathway in dopamine D3receptor mutants during the development of methamphetamine sensitizationJournal of Neurochemistry, 2007
- Role of ERK in cocaine addictionTrends in Neurosciences, 2006
- The Environment versus Genetics in Controlling the Contribution of MAP Kinases to Synaptic PlasticityCurrent Biology, 2006
- The MEK/ERK cascade: From signaling specificity to diverse functionsBiochimica et Biophysica Acta (BBA) - Molecular Cell Research, 2006
- ERK1 and ERK2 mitogen-activated protein kinases affect Ras-dependent cell signaling differentiallyJournal of Biology, 2006
- Knockout of ERK1 Enhances Cocaine-Evoked Immediate Early Gene Expression and Behavioral PlasticityNeuropsychopharmacology, 2006
- DARPP-32 Is a Robust Integrator of Dopamine and Glutamate SignalsPLoS Computational Biology, 2006