Tolerance to competitive NMDA antagonists, but no crosstolerance with barbiturates
- 31 January 1995
- journal article
- Published by Elsevier in Pharmacology Biochemistry and Behavior
- Vol. 50 (1) , 9-15
- https://doi.org/10.1016/0091-3057(94)00215-5
Abstract
No abstract availableKeywords
This publication has 15 references indexed in Scilit:
- Possible involvement of NMDA receptor‐mediated transmission in barbiturate physical dependenceBritish Journal of Pharmacology, 1994
- Chronic administration of NMDA antagonists induces D2 receptor synthesis in rat striatumMolecular Brain Research, 1992
- Down-regulation of cortical β-adrenoceptors by chronic treatment with functional NMDA antagonistsPsychopharmacology, 1992
- Action of chlormethiazole in a model of ethanol withdrawalPsychopharmacology, 1990
- CGP 37849 and CGP 39551: novel and potent competitive N‐methyl‐d‐aspartate receptor antagonists with oral activityBritish Journal of Pharmacology, 1990
- HA-966 antagonizes N-methyl-D-aspartate receptors through a selective interaction with the glycine modulatory siteJournal of Neuroscience, 1989
- Glycine reverses antagonism of N-methyl-D-aspartate (NMDA) by 1-hydroxy-3-aminopyrrolidone-2 (HA-966) but not by D-2-amino-5-phophonovalerate (D-AP5) on rat cortical slicesEuropean Journal of Pharmacology, 1988
- Dissociation of alcohol tolerance and dependenceNature, 1976
- 1‐Hydroxy‐3‐amino‐pyrrolidone‐2 (HA‐966): a new GABA‐like compound, with potential use in extrapyramidal diseasesBritish Journal of Pharmacology, 1971
- Alcohol Dependence Produced in Mice by Inhalation of Ethanol: Grading the Withdrawal ReactionScience, 1971