Activation of SAPK/JNK by camptothecin sensitizes androgen-independent prostate cancer cells to Fas-induced apoptosis
Open Access
- 5 May 2000
- journal article
- Published by Springer Nature in British Journal of Cancer
- Vol. 82 (11) , 1827-1834
- https://doi.org/10.1054/bjoc.2000.1149
Abstract
No abstract availableKeywords
This publication has 47 references indexed in Scilit:
- TRAIL/Apo2L Activates c-Jun NH2-terminal Kinase (JNK) via Caspase-dependent and Caspase-independent PathwaysPublished by Elsevier ,1998
- Signaling by dual specificity kinasesOncogene, 1998
- MEKK1 Binds Directly to the c-Jun N-terminal Kinases/Stress-activated Protein KinasesPublished by Elsevier ,1997
- Fas Induces Cytoplasmic Apoptotic Responses and Activation of the MKK7-JNK/SAPK and MKK6-p38 Pathways Independent of CPP32-like ProteasesThe Journal of cell biology, 1997
- Sequential activation of ICE-like and CPP32-like proteases during Fas-mediated apoptosisNature, 1996
- Fas Ligand-Induced Apoptosis as a Mechanism of Immune PrivilegeScience, 1995
- A c-jun dominant negative mutant protects sympathetic neurons against programmed cell deathNeuron, 1995
- The stress-activated protein kinase subfamily of c-Jun kinasesNature, 1994
- Oncogenic and transcriptional cooperation with Ha-Ras requires phosphorylation of c-Jun on serines 63 and 73Nature, 1991
- A cell-killing monoclonal antibody (anti-Fas) to a cell surface antigen co-downregulated with the receptor of tumor necrosis factor.The Journal of Experimental Medicine, 1989