DNA Methyltransferase 1 Knock Down Induces Gene Expression by a Mechanism Independent of DNA Methylation and Histone Deacetylation
Open Access
- 1 July 2004
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 279 (27) , 27915-27927
- https://doi.org/10.1074/jbc.m312823200
Abstract
No abstract availableKeywords
This publication has 51 references indexed in Scilit:
- Induction of Tumors in Mice by Genomic HypomethylationScience, 2003
- Epigenomic Stress ResponseJournal of Biological Chemistry, 2003
- MCAF Mediates MBD1-Dependent Transcriptional RepressionMolecular and Cellular Biology, 2003
- 5-AzaC Treatment Enhances Expression of Transforming Growth Factor-β Receptors through Down-regulation of Sp3Published by Elsevier ,2001
- Aβ Vasoactivity in VivoAnnals of the New York Academy of Sciences, 2000
- DNA Methyltransferase Inhibition Induces the Transcription of the Tumor Suppressor p21Journal of Biological Chemistry, 2000
- Ras induces p21Cip1/Waf1 cyclin kinase inhibitor transcriptionally through Sp1-binding sitesOncogene, 1999
- Sp1, but not Sp3, functions to mediate promoter activation by TGF-beta through canonical Sp1 binding sitesNucleic Acids Research, 1998
- DNA methylation properties: consequences for pharmacologyTrends in Pharmacological Sciences, 1994
- Targeted mutation of the DNA methyltransferase gene results in embryonic lethalityPublished by Elsevier ,1992