Calmodulin Kinase II and Arrhythmias in a Mouse Model of Cardiac Hypertrophy
- 3 September 2002
- journal article
- research article
- Published by Wolters Kluwer Health in Circulation
- Vol. 106 (10) , 1288-1293
- https://doi.org/10.1161/01.cir.0000027583.73268.e7
Abstract
Background— Calmodulin kinase (CaMK) II is linked to arrhythmia mechanisms in cellular models where repolarization is prolonged. CaMKII upregulation and prolonged repolarization are general feature...Keywords
This publication has 21 references indexed in Scilit:
- The Cardiac-specific Nuclear δB Isoform of Ca2+/Calmodulin-dependent Protein Kinase II Induces Hypertrophy and Dilated Cardiomyopathy Associated with Increased Protein Phosphatase 2A ActivityJournal of Biological Chemistry, 2002
- β-Blockers in Congestive Heart Failure: A Bayesian Meta-AnalysisAnnals of Internal Medicine, 2001
- CaM kinase signaling induces cardiac hypertrophy and activates the MEF2 transcription factor in vivoJournal of Clinical Investigation, 2000
- Differential cellular and subcellular localization of protein phosphatase 1 isoforms in brainJournal of Comparative Neurology, 1999
- Genetic engineering of proteins with cell membrane permeabilityNature Biotechnology, 1998
- The Nuclear δB Isoform of Ca2+/Calmodulin-dependent Protein Kinase II Regulates Atrial Natriuretic Factor Gene Expression in Ventricular MyocytesJournal of Biological Chemistry, 1997
- The Multifunctional Calcium/Calmodulin-Dependent Protein Kinase: From Form to FunctionAnnual Review of Physiology, 1995
- Early afterdepolarizations in cardiac myocytes: mechanism and rate dependenceBiophysical Journal, 1995
- The newly synthesized selective Ca2+calmodulin dependent protein kinase II inhibitor KN-93 reduces dopamine contents in PC12h cellsBiochemical and Biophysical Research Communications, 1991
- Beta-adrenergic stimulation of calcium channels occurs by potentiation of high-activity gating modes.Proceedings of the National Academy of Sciences, 1990