Chronic nicotine treatment counteracts the disappearance of tyrosine-hydroxylase-immunoreactive nerve cell bodies, dendrites and terminals in the mesostriatal dopamine system of the male rat after partial hemitransection
- 1 July 1988
- journal article
- research article
- Published by Elsevier in Brain Research
- Vol. 455 (2) , 332-345
- https://doi.org/10.1016/0006-8993(88)90092-3
Abstract
No abstract availableKeywords
This publication has 30 references indexed in Scilit:
- Partial di-mesencephalic hemitransections produce disappearance of [3H]nicotine binding in discrete regions of rat brainActa Physiologica Scandinavica, 1987
- Increases in dopamine utilization in certain limbic dopamine terminal populations after a short period of intermittent exposure of male rats to cigarette smokeJournal Of Neural Transmission-Parkinsons Disease and Dementia Section, 1986
- Autoradiographic localization of [3H]nicotine binding sites in the rat brainNeuroscience Letters, 1985
- The unbiased estimation of number and sizes of arbitrary particles using the disectorJournal of Microscopy, 1984
- Patterns of cerebral glucose utilization in Parkinson's disease and Huntington's diseaseAnnals of Neurology, 1984
- Smoking and Parkinson's disease.Journal of Neurology, Neurosurgery & Psychiatry, 1982
- Use of avidin-biotin-peroxidase complex (ABC) in immunoperoxidase techniques: a comparison between ABC and unlabeled antibody (PAP) procedures.Journal of Histochemistry & Cytochemistry, 1981
- A new method for receptor autoradiography: [3H]Opioid receptors in rat brainBrain Research, 1979
- A method for simultaneous recording of eight behavioral parameters related to monoamine neurotransmissionPharmacology Biochemistry and Behavior, 1978
- Liquid chromatographic analysis of catecholamines routine assay for regional brain mappingLife Sciences, 1976