Fragile X Mental Retardation Protein Is Required for Synapse Elimination by the Activity-Dependent Transcription Factor MEF2

This publication has 41 references indexed in Scilit:

• Common genetic variants on 5p14.1 associate with autism spectrum disorders
  Nature, 2009
• Autism genome-wide copy number variation reveals ubiquitin and neuronal genes
  Nature, 2009
• Genome-Wide Analysis of MEF2 Transcriptional Program Reveals Synaptic Target Genes and Neuronal Activity-Dependent Polyadenylation Site Selection
  Neuron, 2008
• Fragile X Syndrome: Loss of Local mRNA Regulation Alters Synaptic Development and Function
  Neuron, 2008
• The Self-Tuning Neuron: Synaptic Scaling of Excitatory Synapses
  Cell, 2008
• Cocaine Regulates MEF2 to Control Synaptic and Behavioral Plasticity
  Published by Elsevier ,2008
• Transcription factor MEF2C influences neural stem/progenitor cell differentiation and maturation in vivo
  Proceedings of the National Academy of Sciences, 2008
• MEF2C, a transcription factor that facilitates learning and memory by negative regulation of synapse numbers and function
  Proceedings of the National Academy of Sciences, 2008
• Advances in autism genetics: on the threshold of a new neurobiology
  Nature Reviews Genetics, 2008
• A point mutation in the FMR-1 gene associated with fragile X mental retardation
  Nature Genetics, 1993