ACTH-Responsive, Dexamethasone-Suppressible Adrenocortical Carcinoma

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Abstract
ADRENOCORTICAL neoplasms are not thought to be adrenocorticotrophin (ACTH) dependent since they fail to show suppression of 17-hydroxycorticosteroids (17-OHCS) with the administration of 8 mg of dexamethasone per 24 hours.1 Recently, Kendall and Sloop2 described a patient with an adrenocortical adenoma in whom 2 mg of dexamethasone per 24 hours suppressed urinary 17-OHCS excretion from 18 to 3.5 mg per 24 hours. The following report describes a patient with a metastatic adrenocortical carcinoma that physiologically mimicked adrenocortical hyperplasia.Case ReportEight months after right nephroadrenalectomy for adrenocortical "adenoma" with Cushing's syndrome a 70-year-old woman was admitted to the Clinical Center . . .

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