Degranulation of Natural Killer Cells Following Interaction with HIV-1-Infected Cells Is Hindered by Downmodulation of NTB-A by Vpu
Top Cited Papers
- 1 November 2010
- journal article
- research article
- Published by Elsevier in Cell Host & Microbe
- Vol. 8 (5) , 397-409
- https://doi.org/10.1016/j.chom.2010.10.008
Abstract
No abstract availableKeywords
This publication has 56 references indexed in Scilit:
- Inhibition of lipid antigen presentation in dendritic cells by HIV-1 Vpu interference with CD1d recycling from endosomal compartmentsBlood, 2010
- The Great Escape: Viral Strategies to Counter BST-2/TetherinPLoS Pathogens, 2010
- Minimal requirement for induction of natural cytotoxicity and intersection of activation signals by inhibitory receptorsBlood, 2009
- Vpu Antagonizes BST-2–Mediated Restriction of HIV-1 Release via β-TrCP and Endo-Lysosomal TraffickingPLoS Pathogens, 2009
- Induction of HIV-1 latency and reactivation in primary memory CD4+ T cellsBlood, 2009
- Lysis of Endogenously Infected CD4+ T Cell Blasts by rIL-2 Activated Autologous Natural Killer Cells from HIV-Infected Viremic IndividualsPLoS Pathogens, 2008
- Up on the tightrope: natural killer cell activation and inhibitionNature Immunology, 2008
- The Interferon-Induced Protein BST-2 Restricts HIV-1 Release and Is Downregulated from the Cell Surface by the Viral Vpu ProteinPublished by Elsevier ,2008
- HIV modulates the expression of ligands important in triggering natural killer cell cytotoxic responses on infected primary T-cell blastsBlood, 2007
- HIV-1 Vpr-Induced Apoptosis Is Cell Cycle Dependent and Requires Bax but Not ANTPLoS Pathogens, 2006