Daily cocaine self-administration under long-access conditions augments restraint-induced increases in plasma corticosterone and impairs glucocorticoid receptor-mediated negative feedback in rats

This publication has 58 references indexed in Scilit:

• Restraint-induced corticosterone secretion and hypothalamic CRH mRNA expression are augmented during acute withdrawal from chronic cocaine administration
  Neuroscience Letters, 2007
• Elevation of Glucocorticoids is Necessary but not Sufficient for the Escalation of Cocaine Self-Administration by Chronic Electric Footshock Stress in Rats
  Neuropsychopharmacology, 2006
• Frequency of recent cocaine and alcohol use affects drug craving and associated responses to stress and drug-related cues
  Psychoneuroendocrinology, 2005
• Stress and Cocaine Addiction
  The Journal of Pharmacology and Experimental Therapeutics, 2002
• Transition from Moderate to Excessive Drug Intake: Change in Hedonic Set Point
  Science, 1998
• Diurnal Variations in Plasma ACTH, Cortisol and Beta-Endorphin Levels in Cocaine Addicts
  Hormone Research, 1992
• Neuroendocrine responses to cocaine do not exhibit sensitization following repeated cocaine exposure
  Life Sciences, 1992
• Cocaine stimulates adrenocorticotropin (ACTH) secretion through a corticotropin-releasing factor (CRF)-mediated mechanism
  Brain Research, 1987
• Cocaine induced secretion of ACTH, beta-endorphin, and corticosterone
  Peptides, 1987
• Cleavage of Structural Proteins during the Assembly of the Head of Bacteriophage T4
  Nature, 1970