Inhibition of iNOS gene expression by quercetin is mediated by the inhibition of IκB kinase, nuclear factor-kappa B and STAT1, and depends on heme oxygenase-1 induction in mouse BV-2 microglia
- 1 October 2005
- journal article
- research article
- Published by Elsevier in European Journal of Pharmacology
- Vol. 521 (1-3) , 9-20
- https://doi.org/10.1016/j.ejphar.2005.08.005
Abstract
No abstract availableKeywords
This publication has 51 references indexed in Scilit:
- Profound difference in pharmacokinetics between morin and its isomer quercetin in ratsJournal of Pharmacy and Pharmacology, 2003
- The inhibitory action of quercetin on lipopolysaccharide-induced nitric oxide production in RAW 264.7 macrophage cellsInnate Immunity, 2001
- Epicatechin and its in vivo metabolite, 3′-O-methyl epicatechin, protect human fibroblasts from oxidative-stress-induced cell death involving caspase-3 activationBiochemical Journal, 2001
- Mechanism of Heme Oxygenase-1 Gene Activation by Cadmium in MCF-7 Mammary Epithelial CellsJournal of Biological Chemistry, 2000
- Time course of nitric oxide synthase activity in neuronal, glial, and endothelial cells of rat striatum following focal cerebral ischemiaCellular and Molecular Neurobiology, 1995
- Role of interferon regulatory factor 1 in induction of nitric oxide synthase.The Journal of Experimental Medicine, 1994
- Regulation of the transcription factors NF-κB and AP-1 by redox changesChemico-Biological Interactions, 1994
- Requirement for Transcription Factor IRF-1 in NO Synthase Induction in MacrophagesScience, 1994
- Promoter of the mouse gene encoding calcium-independent nitric oxide synthase confers inducibility by interferon gamma and bacterial lipopolysaccharide.The Journal of Experimental Medicine, 1993
- Microglia and cytokines in neurological disease, with special reference to AIDS and Alzheimer's diseaseGlia, 1993