Histone Acetylation in Keratinocytes Enables Control of the Expression of Cathelicidin and CD14 by 1,25-Dihydroxyvitamin D3

This publication has 31 references indexed in Scilit:

• Inhibition of Histone Deacetylation Promotes Abnormal Epidermal Differentiation and Specifically Suppresses the Expression of the Late Differentiation Marker Profilaggrin
  Journal of Investigative Dermatology, 2007
• Successful treatment of refractory warts with topical vitamin D₃ derivative (maxacalcitol, 1α, 25‐dihydroxy‐22‐oxacalcitriol) in 17 patients
  The Journal of Dermatology, 2007
• Injury enhances TLR2 function and antimicrobial peptide expression through a vitamin D–dependent mechanism
  Journal of Clinical Investigation, 2007
• Wavelength-Dependent Induction of CYP24A1-mRNA after UVB-Triggered Calcitriol Synthesis in Cultured Human Keratinocytes
  Journal of Investigative Dermatology, 2007
• Histone deacetylase activities are required for innate immune cell control of Th1 but not Th2 effector cell function
  Blood, 2006
• Control of the innate epithelial antimicrobial response is cell‐type specific and dependent on relevant microenvironmental stimuli
  Immunology, 2006
• Improved outcome in shigellosis associated with butyrate induction of an endogenous peptide antibiotic
  Proceedings of the National Academy of Sciences, 2006
• Toll-Like Receptor Triggering of a Vitamin D-Mediated Human Antimicrobial Response
  Science, 2006
• Genome-wide Map of Nucleosome Acetylation and Methylation in Yeast
  Published by Elsevier ,2005
• Transforming growth factor-β1 acts cooperatively with sodium n-butyrate to induce differentiation of normal human keratinocytes
  Experimental Cell Research, 1992