Diverse Signaling Pathways Activated by Growth Factor Receptors Induce Broadly Overlapping, Rather Than Independent, Sets of Genes
Open Access
- 1 June 1999
- Vol. 97 (6) , 727-741
- https://doi.org/10.1016/s0092-8674(00)80785-0
Abstract
No abstract availableKeywords
This publication has 55 references indexed in Scilit:
- Bifurcation of Lipid and Protein Kinase Signals of PI3Kγ to the Protein Kinases PKB and MAPKScience, 1998
- STAT3 Serine Phosphorylation by ERK-Dependent and -Independent Pathways Negatively Modulates Its Tyrosine PhosphorylationMolecular and Cellular Biology, 1997
- STATs and Gene RegulationScience, 1997
- CELLULAR RESPONSES TO INTERFERON-γAnnual Review of Immunology, 1997
- Platelet-derived Growth Factor-dependent Cellular Transformation Requires Either Phospholipase Cγ or Phosphatidylinositol 3 KinasePublished by Elsevier ,1997
- Myc but not Fos rescue of PDGF signalling block caused by kinase-inactive SrcNature, 1995
- Requirement for MAP Kinase (ERK2) Activity in Interferon α- and Interferon β-Stimulated Gene Expression Through STAT ProteinsScience, 1995
- Tyr-716 in the platelet-derived growth factor beta-receptor kinase insert is involved in GRB2 binding and Ras activation.Molecular and Cellular Biology, 1994
- PC12 cells overexpressing the insulin receptor undergo insulin-dependent neuronal differentiationCurrent Biology, 1994
- PDGF- and insulin-dependent pp70S6k activation mediated by phosphatidylinositol-3-OH kinaseNature, 1994