Selective retardation of the development of the basal forebrain cholinergic and pontine catecholaminergic nuclei in the brain of trisomy 16 mouse, an animal model of Down's syndrome
- 1 December 1989
- journal article
- research article
- Published by Elsevier in Developmental Brain Research
- Vol. 50 (2) , 251-264
- https://doi.org/10.1016/0165-3806(89)90201-0
Abstract
No abstract availableKeywords
This publication has 37 references indexed in Scilit:
- Neuronal maturation in the foetal brain in Down's syndromeEarly Human Development, 1989
- Down's syndrome and the molecular biology of chromosome 21Progress in Neurobiology, 1988
- Down syndrome, Alzheimer's disease and the trisomy 16 mouseTrends in Neurosciences, 1988
- The neurobiologie consequences of down syndromeBrain Research Bulletin, 1986
- Mouse chimeras composed of trisomy 16 and normal (2n) cells: Preliminary studiesBrain Research Bulletin, 1986
- Mouse Trisomy 16: An Animal Model of Human Trisomy 21 (Down Syndrome)aAnnals of the New York Academy of Sciences, 1985
- Mouse trisomy 16 as an animal model of human trisomy 21 (Down syndrome): Production of viable trisomy 16 ↔ diploid mouse chimerasDevelopmental Biology, 1984
- NGF-Mediated increase of choline acetyltransferase (ChAT) in the neonatal rat forebrain: Evidence for a physiological role of NGF in the brain?Developmental Brain Research, 1983
- Value of an animal model for trisomyVirchows Archiv, 1982
- The development of the septal region in the rat. I. Neurogenesis examined with 3H‐thymidine autoradiographyJournal of Comparative Neurology, 1979