Risperidone-induced action potential prolongation is attenuated by increased repolarization reserve due to concomitant block of ICa,L
- 1 May 2005
- journal article
- Published by Springer Nature in Naunyn-Schmiedebergs Archiv für experimentelle Pathologie und Pharmakologie
- Vol. 371 (5) , 393-400
- https://doi.org/10.1007/s00210-005-1063-5
Abstract
No abstract availableKeywords
This publication has 38 references indexed in Scilit:
- Antiarrhythmic Effects of Ranolazine in a Guinea Pig in Vitro Model of Long-QT SyndromeThe Journal of Pharmacology and Experimental Therapeutics, 2004
- Differential effect of HERG blocking agents on cardiac electrical alternans in the guinea pigEuropean Journal of Pharmacology, 2004
- Block of cardiac delayed‐rectifier and inward‐rectifier K+ currents by nisoldipineBritish Journal of Pharmacology, 2003
- Antipsychotic Drugs: Prolonged QTc Interval, Torsade de Pointes, and Sudden DeathAmerican Journal of Psychiatry, 2001
- Rate dependency of delayed rectifier currents during the guinea‐pig ventricular action potentialThe Journal of Physiology, 2001
- Characterization of Inhibition by Risperidone of the Inwardly Rectifying K+ Current in Pituitary GH3 CellsNeuropsychopharmacology, 2000
- Congenital and acquired long QT syndromeEuropean Heart Journal, 2000
- Frequency-dependent effects of E-4031, almokalant, dofetilide and tedisamil on action potential duration: no evidence for “reverse use dependent” blockNaunyn-Schmiedebergs Archiv für experimentelle Pathologie und Pharmakologie, 1994
- Overdose of risperidoneAnnals of Emergency Medicine, 1993
- Two components of cardiac delayed rectifier K+ current. Differential sensitivity to block by class III antiarrhythmic agents.The Journal of general physiology, 1990