Decay-accelerating factor but not CD59 limits experimental immune-complex glomerulonephritis
Open Access
- 1 April 2007
- journal article
- research article
- Published by Elsevier in Laboratory Investigation
- Vol. 87 (4) , 357-364
- https://doi.org/10.1038/labinvest.3700522
Abstract
No abstract availableKeywords
This publication has 46 references indexed in Scilit:
- CREB transcription factor modulates Bcl2 transcription in response to C5a in HL‐60‐derived neutrophilsEuropean Journal of Clinical Investigation, 2006
- Chemokines and chemokine receptors are involved in the resolution or progression of renal diseaseKidney International, 2003
- Uncontrolled C3 activation causes membranoproliferative glomerulonephritis in mice deficient in complement factor HNature Genetics, 2002
- Glomerular complement regulation is overwhelmed in passive Heymann nephritisKidney International, 2001
- ComplementNew England Journal of Medicine, 2001
- A Role for Mac-1 (CDIIb/CD18) in Immune Complex–stimulated Neutrophil Function In Vivo: Mac-1 Deficiency Abrogates Sustained Fcγ Receptor–dependent Neutrophil Adhesion and Complement-dependent Proteinuria in Acute GlomerulonephritisThe Journal of Experimental Medicine, 1997
- Mouse complement regulatory protein Crry/p65 uses the specific mechanisms of both human decay-accelerating factor and membrane cofactor protein.The Journal of Experimental Medicine, 1995
- Role of CD59 in experimental glomerulonephritis in ratsKidney International, 1994
- Localization of 20-kD homologous restriction factor (HRF20) in diseased human glomeruli. An immunofluorescence studyClinical and Experimental Immunology, 1991
- Immune complex induced glomerular lesions in C5 sufficient and deficient miceKidney International, 1986