Decreased O2 consumption by PMNL from humans and rats with CRF: Role of secondary hyperparathyroidism
- 1 September 1992
- journal article
- Published by Elsevier in Kidney International
- Vol. 42 (3) , 602-609
- https://doi.org/10.1038/ki.1992.324
Abstract
No abstract availableKeywords
This publication has 29 references indexed in Scilit:
- Impaired phagocytosis in chronic renal failure is mediated by secondary hyperparathyroidismKidney International, 1992
- Impaired potassium-induced insulin secretion in chronic renal failureKidney International, 1991
- Inhibition of immunoglobulin production by parathyroid hormone. Implications in chronic renal failureKidney International, 1991
- On the mechanism of impaired insulin secretion in chronic renal failure.Journal of Clinical Investigation, 1991
- Calcium Channels, Stores, and OscillationsAnnual Review of Cell Biology, 1990
- Evidence of Impaired T Cell Function in Hemodialysis Patients: Potential Role for Secondary HyperparathyroidismAmerican Journal of Nephrology, 1990
- Oxidative Metabolism and Phagocytosis of Polymorphonuclear Leukocytes in Patients with Chronic Renal FailureNephron, 1988
- Double stimulation with FMLP and Con A restores the activation of the respiratory burst but not of the phosphoinositide turnover in Ca2+-depleted human neutrophils. A further example of dissociation between stimulation of the NADPH oxidase and phosphoinositide turnoverBiochemical and Biophysical Research Communications, 1986
- Superoxide release by neutrophils: Synergistic effects of a phorbol ester and a calcium ionophoreBiochemical and Biophysical Research Communications, 1984
- Defective Immune and Phagocytic Functions in Uraemia and Renal TransplantationInternational Archives of Allergy and Immunology, 1976