Growth arrest and suppression of tumorigenicity of bladder carcinoma cell lines induced by theP16/CDKN2 (p16INK4A,MTS1) gene and other loci on human chromosome 9
Open Access
- 15 March 1996
- journal article
- research article
- Published by Wiley in International Journal of Cancer
- Vol. 65 (6) , 840-846
- https://doi.org/10.1002/(sici)1097-0215(19960315)65:6<840::aid-ijc22>3.0.co;2-6
Abstract
No abstract availableKeywords
This publication has 14 references indexed in Scilit:
- Fast and sensitive silver staining of DNA in polyacrylamide gelsPublished by Elsevier ,2004
- Frequency of homozygous deletion at p16/CDKN2 in primary human tumoursNature Genetics, 1995
- Diffuse large cell lymphomas exhibit frequent deletions in 9p21–22 and 9q31–34 regionsGenes, Chromosomes and Cancer, 1995
- Frequent somatic mutations and homozygous deletions of the p16 (MTS1) gene in pancreatic adenocarcinomaNature Genetics, 1994
- pl5INK4B is a potentia| effector of TGF-β-induced cell cycle arrestNature, 1994
- Rates of p16 ( MTS1 ) Mutations in Primary Tumors with 9p LossScience, 1994
- A Cell Cycle Regulator Potentially Involved in Genesis of Many Tumor TypesScience, 1994
- Deletions of the cyclin-dependent kinase-4 inhibitor gene in multiple human cancersNature, 1994
- Characterization of chromosome 9 deletions in transitional cell carcinoma by microsatellite assayHuman Molecular Genetics, 1993
- Markers of Neoplastic Transformation in Epithelial Cell Lines Derived From Human CarcinomasJNCI Journal of the National Cancer Institute, 1977