Extracellular adenosine, formed during low level NMDA receptor activation, provides an inhibitory threshold against further NMDA receptor‐midiated neurotransmission in the cortex
- 1 March 1993
- journal article
- research article
- Published by Wiley in Drug Development Research
- Vol. 28 (3) , 406-409
- https://doi.org/10.1002/ddr.430280340
Abstract
No abstract availableKeywords
This publication has 27 references indexed in Scilit:
- N‐Methyl‐d‐Aspartate‐ and Non‐N‐Methyl‐d‐Aspartate‐Evoked Adenosine Release from Rat Cortical Slices: Distinct Purinergic Sources and Mechanisms of ReleaseJournal of Neurochemistry, 1993
- Sites for Antagonism on the N-Methyl-D-Aspartate Receptor Channel ComplexAnnual Review of Pharmacology and Toxicology, 1991
- Purine modulation of cholinomimetic responses in the rat hippocampal sliceBrain Research, 1988
- Long-term potentiation and NMDA receptors in rat visual cortexNature, 1987
- Blockade of "NMDA" Receptors Disrupts Experience-Dependent Plasticity of Kitten Striate CortexScience, 1987
- Excitotoxity and the NMDA receptorTrends in Neurosciences, 1987
- Ketamine—xylazine anaesthesia blocks consolidation of ocular dominance changes in kitten visual cortexNature, 1987
- Adenosine modulation of amygdala kindlingExperimental Neurology, 1984
- Endogenously Released Adenosine Regulates Excitability in the In Vitro HippocampusEpilepsia, 1980
- Adenosine as a putative transmitter in the cerebral cortex. Studies with potentiators and antagonistsLife Sciences, 1975