TGFβ/Smad signaling system and its pathologic correlates

This publication has 103 references indexed in Scilit:

• Polymorphism R25P in the gene encoding transforming growth factor‐beta (TGF‐β₁) is a newly identified risk factor for proliferative diabetic retinopathy
  American Journal of Medical Genetics, 2002
• Role of Transforming Growth Factor β in Human Disease
  New England Journal of Medicine, 2000
• Smad5 Is Essential for Left–Right Asymmetry in Mice
  Developmental Biology, 2000
• Mice lacking Smad3 show accelerated wound healing and an impaired local inflammatory response
  Nature Cell Biology, 1999
• Cellular interpretation of multiple TGF-β signals: intracellular antagonism between activin/BVg1 and BMP-2/4 signaling mediated by Smads
  Development, 1997
• Evidence for a Role of Rho-like GTPases and Stress-activated Protein Kinase/c-Jun N-terminal Kinase (SAPK/JNK) in Transforming Growth Factor β-mediated Signaling
  Published by Elsevier ,1997
• Polymorphisms of the Transforming Growth Factor-β1 Gene in Relation to Myocardial Infarction and Blood Pressure
  Hypertension, 1996
• Overexpression of the c-Myc oncoprotein blocks the growth-inhibitory response but is required for the mitogenic effects of transforming growth factor beta 1.
  Proceedings of the National Academy of Sciences, 1995
• Transforming Growth Factor β as a Predictor of Liver and Lung Fibrosis after Autologous Bone Marrow Transplantation for Advanced Breast Cancer
  New England Journal of Medicine, 1993
• Evolutionary Origins of the Transforming Growth Factor-β Gene Family
  DNA and Cell Biology, 1992