Murine cell complementation of a cold-sensitive defect for simian virus 40 replication in simian cells.
Open Access
- 1 September 1982
- journal article
- research article
- Published by Taylor & Francis in Molecular and Cellular Biology
- Vol. 2 (9) , 1155-1157
- https://doi.org/10.1128/mcb.2.9.1155
Abstract
Mouse 3T3 fibroblasts were found to complement, in simian cell variants semipermissive to simian virus 40, a cold-sensitive defect of an early function, but not a nonconditional defect of viral uncoating. The variant simian cells could rescue simian virus 40 from 3T3 transformants, and this capacity was not temperature dependent.This publication has 5 references indexed in Scilit:
- Cold-sensitive growth of simian virus 40 in semipermissive variants of CV1 cellsJournal of Virology, 1982
- The expression of SV40 large T-antigen in embryonal carcinoma-SV40 transformed somatic cell hybridsVirology, 1980
- Studies on Simian Virus 40 Excision from Cellular ChromosomesCold Spring Harbor Symposia on Quantitative Biology, 1979
- Production of mammalian somatic cell hybrids by means of polyethylene glycol treatmentSomatic Cell and Molecular Genetics, 1975