Mutant Cu,Zn superoxide dismutases and familial amyotrophic lateral sclerosis: evaluation of oxidative hypotheses

This publication has 52 references indexed in Scilit:

• CCS knockout mice establish an alternative source of copper for SOD in ALS
  Free Radical Biology & Medicine, 2002
• Copper, Zinc Superoxide Dismutase and H2O2
  Published by Elsevier ,2002
• Bicarbonate Enhances Peroxidase Activity of Cu,Zn-Superoxide Dismutase
  Journal of Biological Chemistry, 2002
• Superoxide dismutase and the death of motoneurons in ALS
  Trends in Neurosciences, 2001
• Human Cu/Zn Superoxide Dismutase (SOD1) Overexpression in Mice Causes Mitochondrial Vacuolization, Axonal Degeneration, and Premature Motoneuron Death and Accelerates Motoneuron Disease in Mice Expressing a Familial Amyotrophic Lateral Sclerosis Mutant SOD1
  Neurobiology of Disease, 2000
• The Regulation of Mitochondrial Oxygen Uptake by Redox Reactions Involving Nitric Oxide and Ubiquinol
  Journal of Biological Chemistry, 1999
• Epidemiology of mutations in superoxide dismutase in amyotrophic lateal sclerosis
  Annals of Neurology, 1997
• Motor neurons in Cu/Zn superoxide dismutase-deficient mice develop normally but exhibit enhanced cell death after axonal injury
  Nature Genetics, 1996
• The Uniqueness of Superoxide Dismutase (SOD) — Why Cannot Most Copper Compounds Substitute SodIn vivo?
  Free Radical Research Communications, 1988
• Reversal of the superoxide dismutase reaction
  Biochemical and Biophysical Research Communications, 1973