Phosphotyrosine phosphatase activity in the macrophage is enhanced by lipopolysaccharide, tumor necrosis factor α, and granulocyte/macrophage-colony stimulating factor: correlation with priming of the respiratory burst

This publication has 28 references indexed in Scilit:

• Neutrophil priming: the cellular signals that say ‘amber’ but not ‘green’
  Immunology Today, 1995
• Oxygen radicals, nitric oxide and human inflammatory joint disease.
  Annals of the Rheumatic Diseases, 1995
• Activation of the macrophage respiratory burst by phorbol myristate acetate: Evidence for both tyrosine-kinase-dependent and -independent pathways
  Biochimica et Biophysica Acta (BBA) - Molecular Cell Research, 1994
• Dynamin GTPase regulated by protein kinase C phosphorylation in nerve terminals
  Nature, 1993
• Regulation of the p59^fyn protein tyrosine kinase by the CD45 phosphotyrosine phosphatase
  European Journal of Immunology, 1992
• Utility of immobilon-bound phosphoproteins as substrates for protein phosphatases from neutrophils
  Biochimica et Biophysica Acta (BBA) - Molecular Cell Research, 1992
• The superoxide‐generating oxidase of phagocytic cells
  European Journal of Biochemistry, 1991
• Colony stimulating factor-1 Is a negative regulator of the macrophage respiratory burst
  Journal of Cellular Physiology, 1990
• Calyculin A and okadaic acid: Inhibitors of protein phosphatase activity
  Biochemical and Biophysical Research Communications, 1989
• HUMAN PROSTATIC ACID PHOSPHATASE: A HISTIDINE PHOSPHATASE*
  Annals of the New York Academy of Sciences, 1982