Reduced infarct size in the rabbit heartin vivo by ethylisopropyl-amiloride. A role for Na+/H+ exchange
- 1 January 1996
- journal article
- Published by Springer Nature in Basic Research in Cardiology
- Vol. 91 (3) , 203-209
- https://doi.org/10.1007/bf00788906
Abstract
No abstract availableKeywords
This publication has 30 references indexed in Scilit:
- New Na(+)-H+ exchange inhibitor HOE 694 improves postischemic function and high-energy phosphate resynthesis and reduces Ca2+ overload in isolated perfused rabbit heart.Circulation, 1994
- Myocardial phospholipases A2 and their membrane substratesTrends in Cardiovascular Medicine, 1992
- Protective effects of amiloride on the ischemic reperfused rat heart. Relation to mitochondrial functionEuropean Journal of Pharmacology, 1992
- Antiarrhythmic Activity of AmilorideJournal of Cardiovascular Pharmacology, 1991
- Use-Dependent Block of Atrial Sodium Current by EthylisopropylamilorideJournal of Cardiovascular Pharmacology, 1991
- A pH-dependent phospholipase A2 contributes to loss of plasma membrane integrity during chemical hypoxia in rat hepatocytesBiochemical and Biophysical Research Communications, 1991
- Na-H exchange in myocardium: effects of hypoxia and acidification on Na and CaAmerican Journal of Physiology-Cell Physiology, 1990
- Effects of proton buffering and of amiloride derivatives on reperfusion arrhythmias in isolated rat hearts. Possible evidence for an arrhythmogenic role of Na(+)-H+ exchange.Circulation Research, 1990
- The role of the Na+/H+ exchange system in cardiac cells in relation to the control of the internal Na+ concentration. A molecular basis for the antagonistic effect of ouabain and amiloride on the heart.Journal of Biological Chemistry, 1984
- Limitation of myocardial infarct size by atenolol, nadolol and propranolol in dogsEuropean Journal of Pharmacology, 1981