Mouse atonal homolog 1 directs intestinal progenitors to secretory cell rather than absorptive cell fate
- 1 October 2010
- journal article
- Published by Elsevier in Developmental Biology
- Vol. 346 (2) , 215-223
- https://doi.org/10.1016/j.ydbio.2010.07.026
Abstract
No abstract availableKeywords
Funding Information
- Cellular and Molecular Aspects of Systems and Integrative Biology Training Grant (T32-GM08322)
- NIH (PO1-DK06241, RO1-DK56882, RO1-DK78927)
This publication has 33 references indexed in Scilit:
- Hip1r is expressed in gastric parietal cells and is required for tubulovesicle formation and cell survival in miceJournal of Clinical Investigation, 2008
- Loss of intestinal crypt progenitor cells owing to inactivation of both Notch1 and Notch2 is accompanied by derepression of CDK inhibitors p27Kip1 and p57Kip2EMBO Reports, 2008
- Identification of stem cells in small intestine and colon by marker gene Lgr5Nature, 2007
- Hedgehog Signaling in Development and Homeostasis of the Gastrointestinal TractPhysiological Reviews, 2007
- Intestinal Neurogenin 3 directs differentiation of a bipotential secretory progenitor to endocrine cell rather than goblet cell fateDevelopmental Biology, 2007
- Gene expression profiling of gastrin target genes in parietal cellsPhysiological Genomics, 2006
- Gastrointestinal Stem Cells. II. Intestinal stem cellsAmerican Journal of Physiology-Gastrointestinal and Liver Physiology, 2005
- Notch/γ-secretase inhibition turns proliferative cells in intestinal crypts and adenomas into goblet cellsNature, 2005
- Notch signals control the fate of immature progenitor cells in the intestineNature, 2005
- cis Elements of the Villin Gene Control Expression in Restricted Domains of the Vertical (Crypt) and Horizontal (Duodenum, Cecum) Axes of the IntestineJournal of Biological Chemistry, 2002